Q. I'm doing a research project on the food that people with diabetes can eat. The project is a grant proposal, and I need to find out what the consequences are for people with diabetes who make poor food choices are. Do they get sick, can they die, does their blood sugar spike up? Any help would be appreciated. =]
A. Poor food choices lead up to Blindness , Neuropathy , kidney failure , amputations, heart disease ,feeling sick all the time and dying before their time.
1/3 of type1 diabetics die before age 55.
Type 2 diabetics lose up to 13 years of normal life span.
Complications or Sequelae of Diabetes
Sometimes a complication of diabetes may give a clue to the presence of the disease. The principle complications or sequelae associated with diabetes are retinopathy, neuropathy, nephropathy and arteriosclerosis. Whether these are the unavoidable consequences of the diabetic state over time or whether they may be influenced by controlling the diabetes through aggressive monitoring, treatment and life-style management, including diet and supplements, remains a central topic.
One of the largest, most comprehensive diabetes studies conducted to date2 showed that keeping blood sugar levels as close to normal as possible through aggressive management slows the onset and progression of eye, kidney and nerve diseases caused by diabetes. In fact it demonstrated that any sustained lowering of blood sugar helps, even if the person has a history of poor control.
Specifically it found that lowering and maintaining more constant blood sugar levels reduced the risk of eye disease by 76%, kidney disease by 50%, nerve disease by 60% and cardiovascular disease by 35%.
Since the discovery of insulin nearly 70 years ago, the patterns of morbidity from diabetes have changed. Where the major causes of death were ketoacidosis and infection, they are now the microvascular and cardiovascular complications of diabetes (renal failure and myocardial infarction).
These complications are responsible for a reduction in the life expectancy of a newly diagnosed insulin dependent diabetic by about one-third. The basis of managing diabetes in the 90�s is an improvement in the life-style of the diabetic and prevention of complications responsible for morbidity and mortality in diabetes.
Neuropathy (nerve disease)
Diabetic neuropathies are among the most frequent complication of long-term diabetes. It is estimated that 60% to 70% of diabetics have mild to severe forms of nervous system damage. The femoral nerve is commonly involved giving rise to symptoms in the legs and feet. Pain is the chief symptom and tends to worsen at night when the person is at rest. It is usually relieved by activity and aggravated by cold. Paraesthesias are a common accompaniment of the pain. Cramping, tenderness and muscle weakness also occur but atrophy is rare. Advanced femoral nerve disease is a major contributing cause of lower extremity amputations. Nerves in the arms, abdomen and back may also be affected. Symptoms may include impaired heart function, slowed digestion, reduced or absent perspiration, severe oedema, carpal tunnel syndrome, alternating bouts of diarrhoea and constipation, bladder atony, urinary and faecal incontinence and impotence.
With respect to sexual impotence, diabetes is probably the single most common disease associated with erectile failure (termed neurogenic impotence in the diabetic). Since diabetes is a metabolic disease with vascular and nervous system complications and an erection involves all levels of the nervous system from the brain to the peripheral nerves, lesions anywhere along the path may be responsible for erectile failure. It has been estimated that close to 50% of diabetic males have some degree of erectile dysfunction. Neuropathies usually improve with the control of the diabetes. Severe or chronic changes may require several weeks or months to show maximum improvement.
Retinopathy (eye disease)
Changes occurring in the eye which are distinctive of diabetes involve the narrowing, hardening, bulging, haemorrhaging or severing of the veins and capillaries of the retina. This is a serious complication known as retinopathy and may lead to loss of vision. Visual changes in the earlier stages may include diminished vision, contraction of the visual field, changes in the size of objects or photophobia. In the more advanced stage, termed 'proliferative retinopathy', haemorrhages, retinal detachment and other serious forms of deterioration are observed. When the disease progresses to this late stage total blindness may occur.
It usually takes between 10-13 years for diabetic retinopathy to develop and it is present in some degree in most diabetics who have had the disease for 20 years. In only about half of the diabetics who develop it however, is vision markedly impaired and blindness occurs in only about 6%. Still, diabetes is the leading cause of blindness in adults 20 to 74 years old and is estimated to cause from 12,000 to 24,000 new cases each year. Two other complications of diabetes, cataracts and glaucoma, can also lead to loss of vision
And now you know>
1/3 of type1 diabetics die before age 55.
Type 2 diabetics lose up to 13 years of normal life span.
Complications or Sequelae of Diabetes
Sometimes a complication of diabetes may give a clue to the presence of the disease. The principle complications or sequelae associated with diabetes are retinopathy, neuropathy, nephropathy and arteriosclerosis. Whether these are the unavoidable consequences of the diabetic state over time or whether they may be influenced by controlling the diabetes through aggressive monitoring, treatment and life-style management, including diet and supplements, remains a central topic.
One of the largest, most comprehensive diabetes studies conducted to date2 showed that keeping blood sugar levels as close to normal as possible through aggressive management slows the onset and progression of eye, kidney and nerve diseases caused by diabetes. In fact it demonstrated that any sustained lowering of blood sugar helps, even if the person has a history of poor control.
Specifically it found that lowering and maintaining more constant blood sugar levels reduced the risk of eye disease by 76%, kidney disease by 50%, nerve disease by 60% and cardiovascular disease by 35%.
Since the discovery of insulin nearly 70 years ago, the patterns of morbidity from diabetes have changed. Where the major causes of death were ketoacidosis and infection, they are now the microvascular and cardiovascular complications of diabetes (renal failure and myocardial infarction).
These complications are responsible for a reduction in the life expectancy of a newly diagnosed insulin dependent diabetic by about one-third. The basis of managing diabetes in the 90�s is an improvement in the life-style of the diabetic and prevention of complications responsible for morbidity and mortality in diabetes.
Neuropathy (nerve disease)
Diabetic neuropathies are among the most frequent complication of long-term diabetes. It is estimated that 60% to 70% of diabetics have mild to severe forms of nervous system damage. The femoral nerve is commonly involved giving rise to symptoms in the legs and feet. Pain is the chief symptom and tends to worsen at night when the person is at rest. It is usually relieved by activity and aggravated by cold. Paraesthesias are a common accompaniment of the pain. Cramping, tenderness and muscle weakness also occur but atrophy is rare. Advanced femoral nerve disease is a major contributing cause of lower extremity amputations. Nerves in the arms, abdomen and back may also be affected. Symptoms may include impaired heart function, slowed digestion, reduced or absent perspiration, severe oedema, carpal tunnel syndrome, alternating bouts of diarrhoea and constipation, bladder atony, urinary and faecal incontinence and impotence.
With respect to sexual impotence, diabetes is probably the single most common disease associated with erectile failure (termed neurogenic impotence in the diabetic). Since diabetes is a metabolic disease with vascular and nervous system complications and an erection involves all levels of the nervous system from the brain to the peripheral nerves, lesions anywhere along the path may be responsible for erectile failure. It has been estimated that close to 50% of diabetic males have some degree of erectile dysfunction. Neuropathies usually improve with the control of the diabetes. Severe or chronic changes may require several weeks or months to show maximum improvement.
Retinopathy (eye disease)
Changes occurring in the eye which are distinctive of diabetes involve the narrowing, hardening, bulging, haemorrhaging or severing of the veins and capillaries of the retina. This is a serious complication known as retinopathy and may lead to loss of vision. Visual changes in the earlier stages may include diminished vision, contraction of the visual field, changes in the size of objects or photophobia. In the more advanced stage, termed 'proliferative retinopathy', haemorrhages, retinal detachment and other serious forms of deterioration are observed. When the disease progresses to this late stage total blindness may occur.
It usually takes between 10-13 years for diabetic retinopathy to develop and it is present in some degree in most diabetics who have had the disease for 20 years. In only about half of the diabetics who develop it however, is vision markedly impaired and blindness occurs in only about 6%. Still, diabetes is the leading cause of blindness in adults 20 to 74 years old and is estimated to cause from 12,000 to 24,000 new cases each year. Two other complications of diabetes, cataracts and glaucoma, can also lead to loss of vision
And now you know>
Where can I find arguments against an approaching Type 1 diabetes cure?
Q. I'm researching the possibility of cure for Type 1 diabetes, and my argument is that there will be a cure in the near future, if funding improves and if beta cell and immune research continues. This is for my AP research paper, and no, I'm not making others write it. Suprisingly, I can't find a sufficient article arguing that a diabetes cure WON'T come soon, so i'm simply asking, does anyone know where I can find one? Any tips or sites are helpful, thank you!
A. ...as much as I dislike the fact that you are asking yahoo-answers for an AP Essay....I'll help you, my approach would be through stem-cell, maybe grow a healthy pancreas and have a transplant it in, and irradiate your immune system and transplant bone marrow
How to lose weight with type ONE diabetes and hypothyroidism?
Q. Ive been trying extremely hard to lose weight but the scale reads the same number. I have type ONE( not adult on set diabetes you get from poor lifestyle) and hypothyroidism which I think is the problem. I'm so tired of bring in twice the effort as a normal person and not seeing ANYTHING change(not a decimal number, nothing) so does anyone out there know of a diet and exercise plan or trick to help a person with my glitches lose weight? Thanks for the answers!
A. Hey there! I am in the same boat as you. I also have hypothyroidism and type ONE. While weight loss has been excruciatingly hard, I can offer some tips that I have been doing that have helped me slowly but surely get on the fast track to losing some weight.
1. Take a multivitamin: Our bodies are always fighting off so much. If you take a vitamin, you will ensure that your body will be getting the vitamins and minerals it needs to fight off infection and allow your body MORE energy to do other things, such as losing weight. Look for multivitamins that promote a health heart and strong bones, as well as a good immune system.
2. Count your calories: This is hard, but when there is a will, there is a way. Ever look on the nutrition facts of food and note the servings? Follow that! Record your total calories, fat, sodium, etc. Keep a journal and do this. I started out by just recording what I ate in a normal day, evaluating it (weaknesses: eat too big portions, not enough meat, too much bread/sugar, etc) and then seeing what I can do to improve it. Try to keep your calories to about 2,300 per day, and then whittle it down. I'm currently at 2,245 at the most per day, and I'm noting a change in my energy.
3. EXERCISE: Speaking of energy, hypothyroidism simply kills your metabolism with an AK-47. :( RIght here, you just need a little more willpower. Think of living longer. At least that's what I do. Don't use elevators: instead, use the stairs. Walk a little more than you need to. Have Nintendo Wii? Do WiiFit. It works out muscles you never thought you could work out! Walk around your neighborhood every day. Go to a track at a school and walk the curves and jog the straights for thirty minutes to an hour. I totally understand taht your energy will deter you from doing this, but even a little effort should help.
4. Stay happy! SOmething you love to do that doesn't compromise your health or well being? Indulge in it! I like to draw and sew among other things. If you keep your feelings and personality up, then you can do all i have listed above and more.
5. Go to sleep on time. Yes this is very important. Your sleep helps regulate SO much.
While ALL are important, the integral plan is to choose a method and STICK WITH IT! Watch your portions, and work out every day for at least thirty minutes. Take that vitamin, get some sleep, and remember to do something you enjoy.
From one hypobetes dealer to another, I HAVE FAITH IN YOU!
And a last note: I've been doing this very method for the past week and a half, and I lost four pounds. Just keep going, and with a little patience you'll get there!
1. Take a multivitamin: Our bodies are always fighting off so much. If you take a vitamin, you will ensure that your body will be getting the vitamins and minerals it needs to fight off infection and allow your body MORE energy to do other things, such as losing weight. Look for multivitamins that promote a health heart and strong bones, as well as a good immune system.
2. Count your calories: This is hard, but when there is a will, there is a way. Ever look on the nutrition facts of food and note the servings? Follow that! Record your total calories, fat, sodium, etc. Keep a journal and do this. I started out by just recording what I ate in a normal day, evaluating it (weaknesses: eat too big portions, not enough meat, too much bread/sugar, etc) and then seeing what I can do to improve it. Try to keep your calories to about 2,300 per day, and then whittle it down. I'm currently at 2,245 at the most per day, and I'm noting a change in my energy.
3. EXERCISE: Speaking of energy, hypothyroidism simply kills your metabolism with an AK-47. :( RIght here, you just need a little more willpower. Think of living longer. At least that's what I do. Don't use elevators: instead, use the stairs. Walk a little more than you need to. Have Nintendo Wii? Do WiiFit. It works out muscles you never thought you could work out! Walk around your neighborhood every day. Go to a track at a school and walk the curves and jog the straights for thirty minutes to an hour. I totally understand taht your energy will deter you from doing this, but even a little effort should help.
4. Stay happy! SOmething you love to do that doesn't compromise your health or well being? Indulge in it! I like to draw and sew among other things. If you keep your feelings and personality up, then you can do all i have listed above and more.
5. Go to sleep on time. Yes this is very important. Your sleep helps regulate SO much.
While ALL are important, the integral plan is to choose a method and STICK WITH IT! Watch your portions, and work out every day for at least thirty minutes. Take that vitamin, get some sleep, and remember to do something you enjoy.
From one hypobetes dealer to another, I HAVE FAITH IN YOU!
And a last note: I've been doing this very method for the past week and a half, and I lost four pounds. Just keep going, and with a little patience you'll get there!
What is the difference between diabetes insipidus and diabetes mellitus?
Q. i've experienced both extreme thirst and frequent urination, which i know are the symptoms of both insipidus and mellitus. I went to the doctor and he said that i could have either one of those diseases. Im going to do a blood test tomorrow and i was wondering if you guys can give me a few facts on both of these conditions. which condition is more serious? Thanks!
A. DI should not be confused with diabetes mellitus (DM), which results from insulin deficiency or resistance leading to high blood glucose, also called blood sugar. DI and DM are unrelated, although they can have similar signs and symptoms, like excessive thirst and excessive urination.
DM is far more common than DI and receives more news coverage. DM has two main forms, type 1 diabetes and type 2 diabetes. DI is a different form of illness altogether.
Diabetes insipidus (DI) is a rare disease that causes frequent urination and excessive thirst.
DI is not related to diabetes mellitus (DM).
Central DI is caused by damage to the pituitary gland and is treated with a synthetic hormone called desmopressin, which prevents water excretion.
Nephrogenic DI is caused by drugs or kidney disease and is treated with hydrochlorothiazide (HCTZ), indomethacin, or a combination of HCTZ and amiloride.
Scientists have not yet discovered an effective treatment for dipsogenic DI, which is caused by a defect in the thirst mechanism.
Most forms of gestational DI can be treated with desmopressin.
A doctor must determine which type of DI is involved before proper treatment can begin with a water deprivation test, urine test, and/or MRI of the brain (magnetic imaging).
They are both serious conditions that need medical attention. DI can result from a genetic condition in birth that affected the kidneys or a pituitary condition, etc. Always stay hydrated, avoid dehydration with the DI.
Hope this helps.
DM is far more common than DI and receives more news coverage. DM has two main forms, type 1 diabetes and type 2 diabetes. DI is a different form of illness altogether.
Diabetes insipidus (DI) is a rare disease that causes frequent urination and excessive thirst.
DI is not related to diabetes mellitus (DM).
Central DI is caused by damage to the pituitary gland and is treated with a synthetic hormone called desmopressin, which prevents water excretion.
Nephrogenic DI is caused by drugs or kidney disease and is treated with hydrochlorothiazide (HCTZ), indomethacin, or a combination of HCTZ and amiloride.
Scientists have not yet discovered an effective treatment for dipsogenic DI, which is caused by a defect in the thirst mechanism.
Most forms of gestational DI can be treated with desmopressin.
A doctor must determine which type of DI is involved before proper treatment can begin with a water deprivation test, urine test, and/or MRI of the brain (magnetic imaging).
They are both serious conditions that need medical attention. DI can result from a genetic condition in birth that affected the kidneys or a pituitary condition, etc. Always stay hydrated, avoid dehydration with the DI.
Hope this helps.
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